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EJNMMI Research

Table 1 Demographic data

From: Carotid plaque inflammatory activity assessed by 2-[18F]FDG-PET imaging decrease after a neurological thromboembolic event

 

N = 15 patients

Male sex

14 (93%)

Age (years)

71.2 (54–87)

BMI

27.43 (18.65–36.29)

Smoking

 Currently

2 (14%)

 Ever

12 (79%)

Diabetes mellitus

1 (1%)

Hypertension

11 (73%)

History of coronary disease

2 (13%)

COPD

1 (1%)

Previous TIA

1 (1%)

Previous stroke

1 (1%)

Newly diagnosed atrial fibrillation

3 (20%)

Statin lowering treatment*

 Low intensity

8 (53%)

 High intensity

7 (47%)

Statin treatment before current stroke/TIA

6 (40%)

Total cholesterol (mmol/L)

4.78 (2.4–6.7)

LDL cholesterol (mmol/L)

2.72 (0.8–3.5)

HDL cholesterol (mmol/L)

1.50 (0.61–4.7)

Creatinine (µmol/L)

88.00 (55–116)

GFR (mL/min)

72.62 (50–93)

Leucocytes (/L)

7.86 (4.3–10.9)

CRP (mg/L)

5.81 (1–16)

Carotid artery with stenosis degree > 50%

7 (46%)

  1. Data are given in mean and range or percentage in brackets
  2. BMI = body mass index, COPD = chronic obstructive lung disease, TIA = transitory ischemic attack
  3. *Treatment after neurological symptoms. Low intensity defined as Simvastatin ≤ 40 mg or Atorvastatin ≤ 20 mg. High intensity was defined as Simvastatin > 40 mg and Atorvastatin > 20 mg
  4. Blood samples are missing for two patients
  5. Carotid stenosis degree > 50% was defined as a peak systolic velocity > 125 cm/second
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